AMFm should continue.

Cuervo noted, shows that activating the lysosomes of cells – one of the proposed remedies for Huntington’s disease – won’t do any good. ‘It doesn’t matter how active your lysosomes are if they’re not going to receive any cellular parts to digest,’ she said. ‘Instead, we should concentrate on treatments to help autophagosomes recognize intracellular garbage, by minimizing their contact with the defective huntingtin protein perhaps. By enhancing the clearance of cellular debris, we may be able to keep Huntington’s patients free of symptoms for a longer time.